- Considering that the prevalence of epilepsy among autistics is known to be an order of magnitude higher that that of the general population, why is the CDDS-based prevalence of autism rising at a rate of 10% annually (and even higher in the past), whereas the prevalence of epilepsy has always remained at population growth levels?
[John Best Jr. has answered that perhaps epilepsy is not being diagnosed in autistics who have epilepsy. But it's clear in the CDDS data that dual diagnoses are common. He then suggested that mercury does not have a seizure liablity. This, of course, is not consistent with what is known about autism. Plus I've also argued before that an environmental trigger without a seizure liability does not match the numbers. Finally, John asserted that autism and epilepsy are totally unrelated.]
- Why is the prevalence of CDDS clients without mental retardation (presumably based on IQ testing) going up quickly?
- Why is the proportion of autistics with epilepsy going down at an annual rate of -5.5%?
- Why is the proportion of autistics with mental retardation going down at a comparable rate?
- Why do you continue to claim that the number of "new cases" or "newly diagnosed cases" of autism is dropping in California, considering that the CDDS says that they don't provide data on new cases? Isn't it intellectually dishonest to continue to do so?
[I'm not aware of any proponent of the mercury hypothesis having addressed this crucial objection. If you know what the rebuttal consists of, please post it in the comments section.]
- Considering that close to no thimerosal is going into vaccines nowadays, shouldn't the following be true? (1) The number of autistic clients in the CDDS 3-5 age range should be crashing towards zero. (2) All newly diagnosed autistic children would be those vaccinated with thimerosal (via the Flu vaccine, as Sue likes to point out).
- Use of thimerosal has been eliminated or drastically reduced in several countries, including Canada, Denmark and Sweden. Why has no country reported a decrease in he prevalence of autism following these actions?
- By what mechanism does mercury poisoning result in savant skills?
- How does mercury poisoning explain the results of Dawson and Mottron? That is, autistics score higher than the norm in the Raven IQ test. But not only this, the gap between the Weschler and Raven tests is totally the inverse (in the other direction) of what it would be in NTs.
[John Best Jr. proposes that the same gene that results in a mercury excretion liability also produces high IQ. This of course would mean that the mercury spares certain types of cognition. While JB Jr. concedes that genetics could account for cognitive advantages, he does not concede that it's enough to account for socio-linguistic deficits.]
- How does it explain findings by Happe (2001) that parents of autistics have a "cognitive style" (weak central coherence) that can confer information-processing advantages?
- How does mercury poisoning produce significant grey and white matter volume differences in infancy?
[The usual answer to these kinds of questions is that "corrupt scientists" are in on the conspiracy led by the CDC.]
- How does mercury poisoning produce larger brain mass and size?
- How does mercury poisoning produce increased neuron density and smaller neurons?
- Why do you start chelation therapy without first testing for heavy metal poisoning?
- If you have tested your child, did you use a local reputable lab or an internet lab? Did you send a control sample to verify their tests are valid?
- Is there controlled evidence that chelation therapy is an effective treatment for autism?
- Is there controlled evidence that chelation therapy is a safe treatment for the duration you're planning to follow it?
- Are you following medical guidelines as to the proper duration of chelation therapy?
- What is the mercury-based genetic model that explains a 60% concordance for classic autism in identical twins, but a much lower concordance (2%-4%) in siblings and fraternal twins?
- How does mercury poisoning explain a higher proportion of scientists and engineers as close relatives of autistics?
- Why do you propose that autism just cannot be simply genetic, while autism-like conditions such as Fragile-X, Rett Syndrome and Tuberous Sclerosis obviously can?
- How does mercury poisoning explain some of the alleles which have been linked to autism? GABA, SERT, etc.
- Given that autism is more heritable than personality, intelligence, homosexuality and left-handedness, would you say these other variations in human behavior are, too, caused by pathological environmental triggers such as mercury poisoning?
- If you believe that autism did not exist before the 1940s, do you also believe that Down's Syndrome did not exist before 1862, that Fragile-X syndrome did not exist before 1943, and that Rett Syndrome did not exist before 1966?
[JB Jr. admits that Rett Syndrome was likely misdiagnosed as autism before 1966. He further admits that Rett Syndrome might have been called brain damage or birth defect before the 1940s. But when asked if autism could also have been called brain damage or birth defect before the 1940s, he proclaims this is impossible. Why? Because autism did not exist, as Kanner was the first to see it.]
- Do you realize that a prevalence of 1 in 166 today (which includes Asperger's syndrome) cannot be compared to a 1970s or 1980s prevalence?
- Do you understand why anecdotal accounts do not prove an argument?
Characteristics of true heavy metal toxicity
- (Per Jennifer I believe:) If autistics are unable to excrete mercury, wouldn't regular intake of mercury from the environment be enough to kill autistics over time?
- Why are the following symptoms of heavy metal poisoning not characteristic of autism? Headaches, cold hands and feet, vertigo, inability to focus vision, joint and muscle pains, weak pulse, hard pulse, bleeding gums, blisters, tooth ache, jaw inflammation, metallic taste in mouth, loosening of teeth, persistent cough, irregular breathing, swollen lymph nodes in the neck, subnormal temperature, excessive perspiration, chest pain, changes in blood pressure, etc.